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THE 17 WORLD CONGRESS ON PAIN | September 12-16, 2018

Workshops are presented in various formats to provide in-depth review and discussion of cutting edge research, new diagnosis, techniques, and methods, and to answer critical questions in the field.

Workshop formats are described below.

Proposal Basics

The Scientific Program Committee seeks proposals that address topics through a diversity of perspectives. Diversity can mean the approach presented, global composition of a panel, speakers at different stages of their careers, and inclusion of basic science, translational, and clinical perspectives. In its efforts to ensure a well-balanced program, the Scientific Program Committee reserves the right to request changes of specific speakers and/or presentations and to make acceptance of the proposal contingent on those changes.

Special Interest Groups

IASP Special Interest Groups (SIGs) have the opportunity to propose workshops that, if selected, will be scheduled for presentation in the official Congress program. These workshops will be identified in the Congress materials as SIG-sponsored workshops. The Scientific Program Committee will review the proposed workshops for scientific quality in the same manner as for other workshop proposals. Each SIG may propose a maximum of two workshops approved as the official submissions from the SIG. Submission of a workshop by a SIG does not preclude the SIG from submitting a proposal for a Satellite Symposium.

Topical Workshop Speaker Support

Speaking in a Topical Workshop for the world’s largest pain-related gathering is a true honor. IASP values the time and effort that is needed to accomplish such a task and therefore offers complimentary Congress registration to all Workshop Organizers and Speakers accepted to the program. Organizers and Speakers are responsible for all other expenses related to their participation in the Congress, including but not limited to: transportation, hotel accommodations, meals, tips and other incidentals.

Submission Types

Panel Discussions are 90 minute sessions that include a panel of presenters giving in-depth, state-of-the art lectures. Cutting-edge research, clinical treatments, and translation from research to practice are presented. They include a maximum of three speakers, one of whom is also the moderator and session organizer. Three presentations 20 minutes in length each with 5 minutes of questions after are given and approximately 20-30 minutes is left at the end of the session for questions and discussion with the audience to explore common themes from the lectures, challenges addressed, and solutions offered.

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Bringing 4K to a webcam near you

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Have you ever used a webcam and turned up your nose because it can’t push the same amount of pixels as your phone? Well, you’re in luck. Featuring a stunning 2160p resolution and HDR compatibility, the Logitech Brio Webcam has finally brought 4K HDR performance to the webcam world. And if that isn’t enough, it’s also loaded with Windows Hello compatibility and an easy to use stand which will let you position it wherever you need to.

The Webcam of the future

Resolution: 1080p | Features: Background removal, Low-light correction, Windows Cortana and Hello compatibility, Facial tracking

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Creative might not be the first manufacturer that comes to mind when you think of webcams, but after the Creative Labs Senz3D, that might change. This futuristic webcam has more features than you can shake a stick at, and the high-tech 3D scanner and facial tracking tech promises to lead to some intriguing things in both AR functionality and even gaming. It’s expensive, but you really are getting all the bells and whistles with the Creative Labs Senz 3D.

It costs a lot, but it does a lot too

Resolution: 1080p | Features: On-board processing, Zeiss lens, wide angle lens

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The C930e claims to be Logitech's most advanced HD webcam and it's also one of the most expensive ones on the market. But, unlike traditional webcams, which rely on the PC to do the heavy lifting, this unit does the video encoding itself, which should in turn result in better video quality. The wide, 90-degree field of view means it’s well-suited to business videoconferencing and presentations, and of course it’s Skype-certified for PC and Mac.

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Madrigal-Santillán, Eduardo; Bautista, Mirandeli; Gayosso-De-Lucio, Juan A; Reyes-Rosales, Yadira; Posadas-Mondragón, Araceli; Morales-González, Ángel; Soriano-Ursúa, Marvin A; García-Machorro, Jazmín; Madrigal-Bujaidar, Eduardo; Álvarez-González, Isela; Morales-González, José A

AIM: To evaluate the effect of an extract of Geranium schiedeanum (Gs) as a hepatoprotective agent against ethanol (EtOH)-induced toxicity in rats . METHODS: Male Wistar rats weighing 200-230 g were subjected to a 70% partial hepatectomy (PH); they were then divided into three groups (groups 1-3). During the experiment, animals in group 1 drank only water. The other two groups (2-3) drank an aqueous solution of EtOH (40%, v/v). Additionally, rats in group 3 received a Gs extract daily at a dose of 300 mg/kg body weight intragastically. Subsequently, to identify markers of liver damage in serum, alanine aminotransferase, aspartate aminotransferase, albumin and bilirubin were measured by colorimetric methods. Glucose, triglyceride and cholesterol concentrations were also determined. In addition, oxidative damage was estimated by measuring lipid peroxidation [using thiobarbituric-acid reactive substances (TBARS)] in both plasma and the liver and by measuring the total concentration of antioxidants in serum and the total antioxidant capacity in the liver . In addition, a liver mass gain assessment, total DNA analysis and a morpho-histological analysis of the liver from animals in all three groups were performed and compared. Finally, the number of deaths observed in the three groups was analyzed. RESULTS: Administration of the Geranium shiedeanum extract significantly reduced the unfavorable effect of ethanol on liver regeneration (restitution liver mass: PH-EtOH group 60.68% vs PH-Gs-EtOH group 69.22%). This finding was congruent with the reduced levels of hepatic enzymes and the sustained or increased levels of albumin and decreased bilirubin in serum. The extract also modified the metabolic processes that regulate glucose and lipid levels, as observed from the serum measurements. Lower antioxidant levels and the liver damage induced by EtOH administration appeared to be mitigated by the extract, as observed from the TBARs (PH-EtOH group 200.14 mmol/mg vs PH

Mechanism of impaired regeneration of fatty liver in mouse partial hepatectomy model.

Murata, Hiroshi; Yagi, Takahito; Iwagaki, Hiromi; Ogino, Tetsuya; Sadamori, Hiroshi; Matsukawa, Hiroyoshi; Umeda, Yuzoh; Haga, Sanae; Takaka, Noriaki; Ozaki, Michitaka

The mechanism of injury in steatotic liver under pathological conditions been extensively examined. However, the mechanism of an impaired regeneration is still not well understood. The aim of this study was to analyze the mechanism of impaired regeneration of steatotic liver after partial hepatectomy (PH). db/db fatty mice and lean littermates were used for the experiments. Following 70% PH, the survival rate and recovery of liver mass were examined. Liver tissue was histologically examined and analyzed by western blotting and RT-PCR. Of 35 db/db mice, 25 died within 48 h of PH, while all of the control mice survived. Liver regeneration of surviving db/db mice was largely impaired. In db/db mice, mitosis of hepatocytes after PH was disturbed, even though proliferating cell nuclear antigen (PCNA) expression (G1 to S phase marker) in hepatocytes was equally observed in both mice groups. Interestingly, phosphorylation of Cdc2 in db/db mice was suppressed by reduced expression of Wee1 and Myt1, which phosphorylate Cdc2 in S to G2 phase. In steatotic liver , cell-cycle-related proliferative disorders occurred at mid-S phase after PCNA expression. Reduced expression of Wee1 and Myt1 kinases may therefore maintain Cdc2 in an unphosphorylated state and block cell cycle progression in mid-S phase. These kinases may be critical factors involved in the impaired liver regeneration in fatty liver .

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